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PostWysłany: Pon 9:55, 28 Lut 2011    Temat postu: Left ventricular hypertrophy and changes in red bl

,belstaff milano
Red blood cell deformability in left ventricular hypertrophy and mechanism of change


. 'People's Hospital, Rongcheng City,tory burch outlet, Shandong Province. Rong Qi 264,300 postal code for a 1996 article. received a 29 l, l996 for a o7-24 Revised 1996 and -09-18 receive three) nephritis group: 42 cases,belstaff italia, including: acute glomerulonephritis group: A total of lO patients,timberland shoes, 5 males and 5 females down, with the typical clinical symptoms and signs, duration 3.5g/24h,supra shoes outlet, plasma protein <5Og / L. Chronic renal failure group: l2 cases, 6 males and 6 females, of which 5 cases of uremia, azotemia 7 down. 1.2 Methods used adjustable-made SA-B-type viscometer constant pressure capillary whole blood and plasma viscosity (mPa · s). Homemade SA-B-type thrombosis thrombus length was measured (cm), dry, wet weight (mR), determined by ESR Winchester (mm) and hematocrit (), sodium sulfite method with fibrinogen (g / L). Hypertension induced left ventricular hypertrophy possible mechanism. Although ET-I and Ⅱ ET a red blood cell deformability in the two groups were decreased, but only in the ET-I in Chinese patients with hypertension showed significantly higher whole blood viscosity, erythrocyte deformability and thus prompted to decline to a certain extent it can affect whole blood viscosity. Hypertension and the red blood cell deformability was significantly decreased membrane stability mechanism, results from this study with the following factors: (1) contraction of the red cell membrane protein content. Contractile protein membrane proteins account for 35 membrane skeleton, which is an important material to maintain red blood cell morphology, ET group decreased contractile protein of red cell membrane, resulting in significantly increased and MCV shaped red blood cells increases, will inevitably lead to red cell membrane surface area /

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