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<table border="0" cellpadding="0" cellspacing="0" width="100%" class="forumline"> <tr> <td class="cat" width="22%">Autor</td> <td class="cat">Wiadomość</td> </tr> <tr> <td width="22%" align="left" valign="top" class="row1"><span class="name"><a name=""></a><b>nrzxfquxq</b></span></td> <td class="row1" height="28" valign="top"><table width="100%" border="0" cellspacing="0" cellpadding="0"> <tr> <td width="100%"><img src="http://picsrv.fora.pl/Avalanche/images/icon_minipost.gif" width="12" height="9" alt="Post" title="Post" border="0" /><span class="postdetails">Wysłany: Wto 23:53, 08 Mar 2011<span class="gen"> </span> Temat postu: 352 cases of hepatic encephalopathy and prognosis</span></td> </tr> <tr> <td colspan="2"><hr /></td> </tr> <tr> <td colspan="2"><span class="postbody">,<a href="http://www.belstaffoutlets.org" target="_blank" class="postlink">belstaff outlet</a> <br />352 cases of hepatic encephalopathy and prognosis of the incentive <br /> <br /> <br />Incentives for quantity and prognosis Note: 4 groups was significant difference between mortality,<a href="http://www.monclerprezzi.org" target="_blank" class="postlink">Moncler prezzi</a>, P <O. Ol Table 2352 cases of HE stage and prognosis Note: 4 groups was significant difference between mortality, P <O. O13 discussion and the liver cirrhosis and HE is a common complication of severe hepatitis and death, treatment difficulties and high mortality. 352 cases of this group of patients, 73 patients died, mortality was 20.7%, and consistent with CDC and other p】 reported. HE incentives for a variety of common infections, electrolyte imbalance, gastrointestinal bleeding, renal insufficiency, a large number of diuretic, ascites, high-protein diet and drinking. In a variety of incentives, the total of 275 cases of various infections, infection sites of peritoneal infection (256 cases, 63.6%), pulmonary infection (83 cases, 23.6%), the most common, indicating that the infection has become the most HE the main incentives consistent with the reported 141 domestic. Cellular immunity in patients with severe liver disease and humoral immune function, intestinal barrier dysfunction, portosystemic shunt formation, such as hypersplenism, liver cells to bacterial and viral clearance decline, complicated by a variety of infections. Infection, increased body catabolism, ammonia production increased, but also the liver, brain damage to the real organ, the brain oxygen and heat sensitivity of the toxicity of ammonia also increased significantly, can induce HE. 224 cases of electrolyte disturbance, most of the low sodium, low potassium. Patients with advanced liver and kidney syndrome, liver disease, liver damage, decreased albumin synthesis, plasma colloid osmotic pressure decreased, causing leakage of tissue fluid. Diuretic with a strong diuretic, ascites, inadequate intake, salt restriction limited water, nausea, vomiting and diarrhea, can be manifested as hyponatremia, hypokalemia. Easily lead to hypokalemia hypokalemic alkalosis, an increase in free ammonia in blood, the blood brain barrier into the brain tissue, induced HE. HE is very common when the base hyperlipidemia, often leading cause of death of patients. Therefore, in the rescue process, special attention should be prevention and treatment of hyperlipidemia base. Hyponatremia accelerate tissue decomposition, so that decomposition products of urea increased intracellular osmotic pressure can affect the result of cerebral edema, induced HE. Ll0 cases of upper gastrointestinal bleeding. Intestinal bleeding in the blood produced by the bacterial decomposition of ammonia is absorbed through the blood-brain barrier leading to brain dysfunction; the other hand, bleeding body to reduce blood volume, affecting brain function, but also increase liver ischemia, lack of oxygen and liver damage, the liver clearance of ammonia reduced the elevated blood ammonia, induced HE. 95 cases of renal insufficiency. Renal insufficiency can lead to reduction of ammonia and other toxins removal, water and electrolyte balance disorders, induce and aggravate HE. Renal dysfunction and liver cirrhosis and hyponatremia is an important complication of severe hepatitis is a common incentive for HE, in particular, hepatorenal syndrome, the treatment is poor, high mortality I31. L1 large number of cases of diuretic paracentesis. Ascites, large amounts of protein loss, increased liver damage, thus enabling the absorption of ammonia and through blood-brain barrier. Long-term heavy use of diuretics, to electrolyte imbalance, acid-base imbalance, induced HE. Inappropriate diet, high protein diet in 9 cases. When liver cells are damaged, poor metabolism of proteins, if the intake of high protein diet, without the digestion and absorption of protein in the intestine by bacterial decomposition, produce large amounts of ammonia induced by HE. 8 cases of long-term heavy drinking. Liver dysfunction in patients with liver disease, detoxification capacity decreased, drinking lead to liver cell necrosis caused by more HE. HE high mortality and poor prognosis, often the result of a variety of incentives to work together. The group data show that mortality increased with the incentive to rise, the more incentive, the higher mortality, consistent with national studies 141. In addition, HE the higher stages, the higher the mortality. The complication with poor prognosis. The above analysis that, HE is a clinical Intensive advanced liver disease, the number of outcomes and incentives, HE staging, complications are closely related. Therefore, early diagnosis of HE, and actively prevent and remove all kinds of incentives and complications, is to reduce the occurrence of HE and improve the prognosis.</span></td> </tr> </table></td> </tr> <tr> <td colspan="2" height="1" class="spaceRow"><img src="http://picsrv.fora.pl/Avalanche/images/spacer.gif" alt="" width="1" height="1" /></td> </tr> </table>
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